electroencephalography system Search Results


94
NeuroNexus Technologies eeg array
Eeg Array, supplied by NeuroNexus Technologies, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Welcony net amps 300 amplifier
Net Amps 300 Amplifier, supplied by Welcony, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ADInstruments diameter shielded wires
Diameter Shielded Wires, supplied by ADInstruments, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ADInstruments ag agcl sintered electrodes
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ADInstruments electroencephalography eeg
Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative <t>EEG</t> re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.
Electroencephalography Eeg, supplied by ADInstruments, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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PLUX Biosignals SA eeg sensor
Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative <t>EEG</t> re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.
Eeg Sensor, supplied by PLUX Biosignals SA, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
Wolters Kluwer Health electroencephalography: basic principles, clinical applications, and related fields 5th edn
Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative <t>EEG</t> re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.
Electroencephalography: Basic Principles, Clinical Applications, And Related Fields 5th Edn, supplied by Wolters Kluwer Health, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
BioSemi 248-channel electroencephalography
Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative <t>EEG</t> re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.
248 Channel Electroencephalography, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
Easycap gmbh eeg-1200
VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior <t>electrodes,</t> particularly at Oz
Eeg 1200, supplied by Easycap gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
brain products gmbh portable 64-channel eeg system liveamp 64
VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior <t>electrodes,</t> particularly at Oz
Portable 64 Channel Eeg System Liveamp 64, supplied by brain products gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Neuralynx inc eeg electroencephalography
VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior <t>electrodes,</t> particularly at Oz
Eeg Electroencephalography, supplied by Neuralynx inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Compumedics Neuroscan syn-amps electroencephalography (eeg) amplifier
VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior <t>electrodes,</t> particularly at Oz
Syn Amps Electroencephalography (Eeg) Amplifier, supplied by Compumedics Neuroscan, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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Image Search Results


Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative EEG re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.

Journal: Stroke

Article Title: Pharmacological Induction of Heme Oxygenase-1 by a Triterpenoid Protects Neurons Against Ischemic Injury

doi: 10.1161/strokeaha.111.647420

Figure Lengend Snippet: Figure 5. CDDO-Im protects hippocampal CA1 neurons against global ischemia via HO-1 in rats. A, A representative EEG re- cording in a rat undergoing global cerebral ischemia, showing rapid isoelectricity after the onset of ischemia. B, H&E stain of CA1 regions and quantitative analysis of viable neurons after global ischemia in rats. a, sham; (b) vehicle-treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and tin protoporphyrin IX (Sn- PPIX)-treated ischemia. Bar50 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. C, PANT stain of CA1 regions and quantitative anal- ysis of dead neurons. a, sham; (b) vehicle- treated ischemia; (c) CDDO-Im-treated ischemia; and (d) CDDO-Im- and Sn-PPIX- treated ischemia. Bar100 m; n8, *P0.05 versus sham, #P0.05 versus vehicle-treated ischemia, and &P0.05 versus CDDO-treated ischemia. D, West- ern blots of HO-1 in CA1, demonstrating pre-existence of HO-1 in CA1 after CDDO-Im treatments. n3, **P0.01 ver- sus sham, ##P0.01 versus sham and vehicle-treated ischemia. S indicates sham; V, vehicle-treated; CD, CDDO-Im- treated; CDDO-Im, 2-cyano-3,12 dioxooleana-1,9 dien-28-oyl imidazoline; HO-1, heme oxygenase-1; EEG, electroen- cephalography; H&E, hematoxylin and eosin.

Article Snippet: Electroencephalography (EEG) was recorded to ensure isoelectricity at the onset of ischemia using a PowerLab system (ADInstruments, Colorado Springs, CO).

Techniques: Staining

VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior electrodes, particularly at Oz

Journal: Cognitive Neurodynamics

Article Title: Involvement of the visual change detection process in facilitating perceptual alternation in the bistable image

doi: 10.1007/s11571-017-9430-8

Figure Lengend Snippet: VEPs to the target image for DEV and STD targets. a VEPs at POz and Oz are shown for the DEV and STD targets, respectively. The VEP amplitude for the DEV target was significantly higher than that for the STD target. The time interval in which there was a significant difference is shaded in gray (for the procedure of the statistical analysis, see the “Methods” section). b Isocontour maps at latencies of 150 and 300 ms are shown for DEV and STD targets. In both latencies, VEP was negatively enhanced in amplitude at the posterior electrodes, particularly at Oz

Article Snippet: Neural activity in the DEV and STD conditions was recorded by an electroencephalography (EEG) processor with 57 electrodes (EEG-1200, Nihon Kohden, Tokyo, Japan; EasyCap GmbH, Herrsching, Germany).

Techniques: